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What is the proposed mechanism behind BPC-157 and tendon healing in the published literature?

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The published literature reports that BPC-157 appears to promote tendon healing largely through angiogenic and fibroblast-directed pathways. In animal models, investigators have associated it with upregulation of the VEGFR2 receptor and increased outgrowth and migration of tendon fibroblasts, alongside modulation of the nitric oxide system [1]. These are preclinical, mechanistic findings, so they describe what has been observed in research settings rather than any established clinical effect.

Sources: [1] BPC-157 monograph

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